Presenilin1 exerts antiproliferative effects by repressing the Wnt/β-catenin pathway in glioblastoma

BACKGROUND: Glioblastoma and Alzheimer’s disease (AD) are the most common and devastating diseases in the central nervous system. The dysfunction of Presenilin1 is the main reason for AD pathogenesis. However, the molecular function of Presenilin1 and its relative mechanism in glioblastoma remain un...

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Detalles Bibliográficos
Autores principales: Yang, Wei, Wu, Peng-fei, Ma, Jian-xing, Liao, Mao-jun, Xu, Lun-shan, Xu, Min-hui, Yi, Liang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Methodology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7014622/
https://www.ncbi.nlm.nih.gov/pubmed/32046730
http://dx.doi.org/10.1186/s12964-019-0501-9

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7014622/
https://www.ncbi.nlm.nih.gov/pubmed/32046730
http://dx.doi.org/10.1186/s12964-019-0501-9

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