Reciprocal Regulatory Interaction between TRPV1 and Kinin B1 Receptor in a Rat Neuropathic Pain Model

Kinins are mediators of pain and inflammation and evidence suggests that the inducible kinin B1 receptor (B1R) is involved in neuropathic pain (NP). This study investigates whether B1R and TRPV1 are colocalized on nociceptors and/or astrocytes to enable regulatory interaction either directly or thro...

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Detalles Bibliográficos
Autores principales: Cernit, Veronica, Sénécal, Jacques, Othman, Rahmeh, Couture, Réjean
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7037982/
https://www.ncbi.nlm.nih.gov/pubmed/32012798
http://dx.doi.org/10.3390/ijms21030821

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7037982/
https://www.ncbi.nlm.nih.gov/pubmed/32012798
http://dx.doi.org/10.3390/ijms21030821

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