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Helicobacter pylori infection leads to KLF4 inactivation in gastric cancer through a TET1‐mediated DNA methylation mechanism
Krüppel‐like factor 4 (KLF4) has a tumor suppressor role in the progression of gastric cancer (GC), and inhibition or loss of KLF4 expression was identified in GC. The aim of this study was to explore the new molecular mechanism of KLF4 inactivation in gastric cancer. Herein, we report that Helicoba...
Autores principales: | Zhao, Rongrong, Liu, Zhengxia, Xu, Wenting, Song, Le, Ren, Haifeng, Ou, Yang, Liu, Yakun, Wang, Siying |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7131848/ https://www.ncbi.nlm.nih.gov/pubmed/32017451 http://dx.doi.org/10.1002/cam4.2892 |
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Correction to Helicobacter pylori infection leads to KLF4 inactivation in gastric cancer through a TET1‐mediated DNA methylation mechanismZhao, R, Liu, Z, Xu, W, Song L., Ren H., Ou Y., Liu Y., Wang S. Helicobacter pylori infection leads to KLF4 inactivation in gastric cancer through a TET1‐mediated DNA methylation mechanism. Cancer Med 2020; 9: 2551–2563. https://doi.org/10.1002/cam4.2892
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