Prevention of Doxorubicin-Induced Autophagy Attenuates Oxidative Stress and Skeletal Muscle Dysfunction

Clinical use of the chemotherapeutic doxorubicin (DOX) promotes skeletal muscle atrophy and weakness, adversely affecting patient mobility and strength. Although the mechanisms responsible for DOX-induced skeletal muscle dysfunction remain unclear, studies implicate the significant production of rea...

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Detalles Bibliográficos
Autores principales: Doerr, Vivian, Montalvo, Ryan N., Kwon, Oh Sung, Talbert, Erin E., Hain, Brian A., Houston, Fraser E., Smuder, Ashley J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7139604/
https://www.ncbi.nlm.nih.gov/pubmed/32210013
http://dx.doi.org/10.3390/antiox9030263

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7139604/
https://www.ncbi.nlm.nih.gov/pubmed/32210013
http://dx.doi.org/10.3390/antiox9030263

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