Increase in PKCα Activity during Heart Failure Despite the Stimulation of PKCα Braking Mechanism

Rationale: Heart failure (HF) is marked by dampened cardiac contractility. A mild therapeutic target that improves contractile function without desensitizing the β-adrenergic system during HF may improve cardiac contractility and potentially survival. Inhibiting protein kinase C α (PKCα) activity ma...

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Detalles Bibliográficos
Autor principal: Aslam, Naveed
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7177253/
https://www.ncbi.nlm.nih.gov/pubmed/32272716
http://dx.doi.org/10.3390/ijms21072561

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7177253/
https://www.ncbi.nlm.nih.gov/pubmed/32272716
http://dx.doi.org/10.3390/ijms21072561

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