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Increase in PKCα Activity during Heart Failure Despite the Stimulation of PKCα Braking Mechanism
Rationale: Heart failure (HF) is marked by dampened cardiac contractility. A mild therapeutic target that improves contractile function without desensitizing the β-adrenergic system during HF may improve cardiac contractility and potentially survival. Inhibiting protein kinase C α (PKCα) activity ma...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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MDPI
2020
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7177253/ https://www.ncbi.nlm.nih.gov/pubmed/32272716 http://dx.doi.org/10.3390/ijms21072561 |