Inhibition of formyl peptide receptors improves the outcome in a mouse model of Alzheimer disease

BACKGROUND: An important hallmark of Alzheimer’s disease (AD) is the increase of Aβ1-42 burden and its accumulation to senile plaques, leading the reactive gliosis and neurodegeneration. The modulation of glia cell function represents an attractive therapeutic strategy, but is currently limited by a...

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Detalles Bibliográficos
Autores principales: Schröder, Nicole, Schaffrath, Anja, Welter, Josua A., Putzka, Tim, Griep, Angelika, Ziegler, Patrick, Brandt, Elisa, Samer, Sebastian, Heneka, Michael T., Kaddatz, Hannes, Zhan, Jiangshan, Kipp, Eugenia, Pufe, Thomas, Tauber, Simone C., Kipp, Markus, Brandenburg, Lars-Ove
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7181500/
https://www.ncbi.nlm.nih.gov/pubmed/32331524
http://dx.doi.org/10.1186/s12974-020-01816-2

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7181500/
https://www.ncbi.nlm.nih.gov/pubmed/32331524
http://dx.doi.org/10.1186/s12974-020-01816-2

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