A Novel Zinc Chelator, 1H10, Ameliorates Experimental Autoimmune Encephalomyelitis by Modulating Zinc Toxicity and AMPK Activation

Previous studies in our lab revealed that chemical zinc chelation or zinc transporter 3 (ZnT3) gene deletion suppresses the clinical features and neuropathological changes associated with experimental autoimmune encephalomyelitis (EAE). In addition, although protective functions are well documented...

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Detalles Bibliográficos
Autores principales: Choi, Bo Young, Jeong, Jeong Hyun, Eom, Jae-Won, Koh, Jae-Young, Kim, Yang-Hee, Suh, Sang Won
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7247014/
https://www.ncbi.nlm.nih.gov/pubmed/32397660
http://dx.doi.org/10.3390/ijms21093375

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7247014/
https://www.ncbi.nlm.nih.gov/pubmed/32397660
http://dx.doi.org/10.3390/ijms21093375

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