Cargando…
Entry and exit of chemotherapeutically-promoted cellular dormancy in glioblastoma cells is differentially affected by the chemokines CXCL12, CXCL16, and CX3CL1
Glioblastoma multiforme (GBM) is a malignant brain tumor that evades therapy regimens. Since cellular dormancy is one strategy for surviving, and since chemokines determine the environmental conditions in which dormancy occurs, we investigated how chemokines affect temozolomide (TMZ)-promoted cellul...
Autores principales: | Adamski, Vivian, Hattermann, Kirsten, Kubelt, Carolin, Cohrs, Gesa, Lucius, Ralph, Synowitz, Michael, Sebens, Susanne, Held-Feindt, Janka |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7253351/ https://www.ncbi.nlm.nih.gov/pubmed/32346064 http://dx.doi.org/10.1038/s41388-020-1302-8 |
Ejemplares similares
-
The Chemokine Receptor CXCR6 Evokes Reverse Signaling via the Transmembrane Chemokine CXCL16
por: Adamski, Vivian, et al.
Publicado: (2017) -
Effects of the chemokine CXCL12 and combined internalization of its receptors CXCR4 and CXCR7 in human MCF-7 breast cancer cells
por: Hattermann, Kirsten, et al.
Publicado: (2014) -
“Inverse signaling” of the transmembrane chemokine CXCL16 contributes to proliferative and anti-apoptotic effects in cultured human meningioma cells
por: Hattermann, Kirsten, et al.
Publicado: (2016) -
Insights into Gene Regulation under Temozolomide-Promoted Cellular Dormancy and Its Connection to Stemness in Human Glioblastoma
por: Kubelt, Carolin, et al.
Publicado: (2023) -
Influence of Simulated Deep Brain Stimulation on the Expression of Inflammatory Mediators by Human Central Nervous System Cells In Vitro
por: Kubelt, Carolin, et al.
Publicado: (2021)