Identification of Clec4b as a novel regulator of bystander activation of auto-reactive T cells and autoimmune disease

The control of chronic inflammation is dependent on the possibility of limiting bystander activation of autoreactive and potentially pathogenic T cells. We have identified a non-sense loss of function single nucleotide polymorphism in the C-type lectin receptor, Clec4b, and have shown that it contro...

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Detalles Bibliográficos
Autores principales: Bäckdahl, Liselotte, Aoun, Mike, Norin, Ulrika, Holmdahl, Rikard
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2020
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7297379/
https://www.ncbi.nlm.nih.gov/pubmed/32497089
http://dx.doi.org/10.1371/journal.pgen.1008788

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7297379/
https://www.ncbi.nlm.nih.gov/pubmed/32497089
http://dx.doi.org/10.1371/journal.pgen.1008788

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