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Identification of Clec4b as a novel regulator of bystander activation of auto-reactive T cells and autoimmune disease
The control of chronic inflammation is dependent on the possibility of limiting bystander activation of autoreactive and potentially pathogenic T cells. We have identified a non-sense loss of function single nucleotide polymorphism in the C-type lectin receptor, Clec4b, and have shown that it contro...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Public Library of Science
2020
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7297379/ https://www.ncbi.nlm.nih.gov/pubmed/32497089 http://dx.doi.org/10.1371/journal.pgen.1008788 |