Oxidative DNA damage is concurrently repaired by base excision repair (BER) and apyrimidinic endonuclease 1 (APE1)‐initiated nonhomologous end joining (NHEJ) in cortical neurons

AIMS: Accumulating studies have suggested that base excision repair (BER) is the major repair pathway of oxidative DNA damage in neurons, and neurons are deficient in other DNA repair pathways, including nucleotide excision repair and homologous recombination repair. However, some studies have demon...

Descripción completa

Detalles Bibliográficos
Autores principales: Yang, J.‐L., Chen, W.‐Y., Mukda, S., Yang, Y.‐R., Sun, S.‐F., Chen, S.‐D.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2019
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7317839/
https://www.ncbi.nlm.nih.gov/pubmed/31628877
http://dx.doi.org/10.1111/nan.12584

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7317839/
https://www.ncbi.nlm.nih.gov/pubmed/31628877
http://dx.doi.org/10.1111/nan.12584

Ejemplares similares