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Oxidative DNA damage is concurrently repaired by base excision repair (BER) and apyrimidinic endonuclease 1 (APE1)‐initiated nonhomologous end joining (NHEJ) in cortical neurons
AIMS: Accumulating studies have suggested that base excision repair (BER) is the major repair pathway of oxidative DNA damage in neurons, and neurons are deficient in other DNA repair pathways, including nucleotide excision repair and homologous recombination repair. However, some studies have demon...
Autores principales: | Yang, J.‐L., Chen, W.‐Y., Mukda, S., Yang, Y.‐R., Sun, S.‐F., Chen, S.‐D. |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2019
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7317839/ https://www.ncbi.nlm.nih.gov/pubmed/31628877 http://dx.doi.org/10.1111/nan.12584 |
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