Low levels of AMPK promote epithelial‐mesenchymal transition in lung cancer primarily through HDAC4‐ and HDAC5‐mediated metabolic reprogramming

AMP‐activated protein kinase (AMPK) serves as a “supermetabolic regulator” that helps maintain cellular energy homeostasis. However, the role of AMPK in glucose metabolism reprogramming in lung cancer remains unclear. Here, our study shows that low AMPK expression correlates with metastasis and clin...

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Detalles Bibliográficos
Autores principales: Feng, Shoujie, Zhang, Li, Liu, Xiucheng, Li, Guangbin, Zhang, Biao, Wang, Ziwen, Zhang, Hao, Ma, Haitao
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2020
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7348170/
https://www.ncbi.nlm.nih.gov/pubmed/32519437
http://dx.doi.org/10.1111/jcmm.15410

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7348170/
https://www.ncbi.nlm.nih.gov/pubmed/32519437
http://dx.doi.org/10.1111/jcmm.15410

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