Catalpol ameliorates doxorubicin-induced inflammation and oxidative stress in H9C2 cells through PPAR-γ activation

Drug-induced cardiomyopathy is a severe disease that leads to refractory heart disease at late stages, with increasing detrimental effects. DOX-induced cell damage is primarily induced via cellular oxidative stress. The present study investigated the effects of catalpol on doxorubicin (DOX)-induced...

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Detalles Bibliográficos
Autores principales: Jiang, Yanjie, Zhang, Qing
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2020
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7388568/
https://www.ncbi.nlm.nih.gov/pubmed/32765656
http://dx.doi.org/10.3892/etm.2020.8743

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7388568/
https://www.ncbi.nlm.nih.gov/pubmed/32765656
http://dx.doi.org/10.3892/etm.2020.8743

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