Synaptic Plasticity After Focal Cerebral Ischemia Was Attenuated by Gap26 but Enhanced by GAP-134

Objective: Synaptic plasticity is critical for neurorehabilitation after focal cerebral ischemia. Connexin 43 (Cx43), the main component of the gap junction, has been shown to be pivotal for synaptic plasticity. The objective of this study was to investigate the role of the Cx43 inhibitor (Gap26) an...

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Detalles Bibliográficos
Autores principales: Yang, Kailing, Zhou, Ying, Zhou, Lequan, Yan, Fuman, Guan, Li, Liu, Haimei, Liu, Wei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2020
Materias:
Neurology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7479336/
https://www.ncbi.nlm.nih.gov/pubmed/32982919
http://dx.doi.org/10.3389/fneur.2020.00888

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7479336/
https://www.ncbi.nlm.nih.gov/pubmed/32982919
http://dx.doi.org/10.3389/fneur.2020.00888

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