S100B Impairs Oligodendrogenesis and Myelin Repair Following Demyelination Through RAGE Engagement

Increased expression of S100B and its specific receptor for advanced glycation end products (RAGE) has been described in patients with multiple sclerosis (MS), being associated with an active demyelinating process. We previously showed that a direct neutralization of S100B reduces lysophosphatidylch...

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Detalles Bibliográficos
Autores principales: Santos, Gisela, Barateiro, Andreia, Brites, Dora, Fernandes, Adelaide
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2020
Materias:
Cellular Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7500156/
https://www.ncbi.nlm.nih.gov/pubmed/33100970
http://dx.doi.org/10.3389/fncel.2020.00279

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7500156/
https://www.ncbi.nlm.nih.gov/pubmed/33100970
http://dx.doi.org/10.3389/fncel.2020.00279

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