Neutralization of Lipocalin-2 Diminishes Stroke-Reperfusion Injury

Oxidative stress is a key contributor to the pathogenesis of stroke-reperfusion injury. Neuroinflammatory peptides released after ischemic stroke mediate reperfusion injury. Previous studies, including ours, have shown that lipocalin-2 (LCN2) is secreted in response to cerebral ischemia to promote r...

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Detalles Bibliográficos
Autores principales: Wang, Guona, Weng, Yi-Chinn, Chiang, I-Chen, Huang, Yu-Ting, Liao, Yi-Chu, Chen, Yi-Chun, Kao, Cheng-Yuan, Liu, Yu-Li, Lee, Tsong-Hai, Chou, Wen-Hai
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7503651/
https://www.ncbi.nlm.nih.gov/pubmed/32872405
http://dx.doi.org/10.3390/ijms21176253

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7503651/
https://www.ncbi.nlm.nih.gov/pubmed/32872405
http://dx.doi.org/10.3390/ijms21176253

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