GPR68 deletion impairs hippocampal long-term potentiation and passive avoidance behavior

Increased neural activities reduced pH at the synaptic cleft and interstitial spaces. Recent studies have shown that protons function as a neurotransmitter. However, it remains unclear whether protons signal through a metabotropic receptor to regulate synaptic function. Here, we showed that GPR68, a...

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Detalles Bibliográficos
Autores principales: Xu, Yuanyuan, Lin, Mike T., Zha, Xiang-ming
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Micro Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7526169/
https://www.ncbi.nlm.nih.gov/pubmed/32993733
http://dx.doi.org/10.1186/s13041-020-00672-8

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7526169/
https://www.ncbi.nlm.nih.gov/pubmed/32993733
http://dx.doi.org/10.1186/s13041-020-00672-8

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