Cardiomyocyte-derived small extracellular vesicles can signal eNOS activation in cardiac microvascular endothelial cells to protect against Ischemia/Reperfusion injury

Rationale: The crosstalk between cardiac microvascular endothelial cells (CMECs) and cardiomyocytes (CMs) has emerged as a key component in the development of, and protection against, cardiac diseases. For example, activation of endothelial nitric oxide synthase (eNOS) in CMECs, by therapeutic strat...

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Detalles Bibliográficos
Autores principales: Chen, Guihao, Xu, Chuansheng, Gillette, Thomas G., Huang, Tongyi, Huang, Peisen, Li, Qing, Li, Xiangdong, Li, Qinfeng, Ning, Yu, Tang, Ruijie, Huang, Cunrong, Xiong, Yuyan, Tian, Xiaqiu, Xu, Jun, Xu, Junyan, Chang, Liping, Wei, Cong, Jin, Chen, Hill, Joseph A., Yang, Yuejin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Ivyspring International Publisher 2020
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7546010/
https://www.ncbi.nlm.nih.gov/pubmed/33052245
http://dx.doi.org/10.7150/thno.43163