Molecular and Cellular Substrates for the Friedreich Ataxia. Significance of Contactin Expression and of Antioxidant Administration

In this study, the neural phenotype is explored in rodent models of the spinocerebellar disorder known as the Friedreich Ataxia (FA), which results from mutations within the gene encoding the Frataxin mitochondrial protein. For this, the M12 line, bearing a targeted mutation, which disrupts the Frat...

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Detalles Bibliográficos
Autores principales: Bizzoca, Antonella, Caracciolo, Martina, Corsi, Patrizia, Magrone, Thea, Jirillo, Emilio, Gennarini, Gianfranco
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7570916/
https://www.ncbi.nlm.nih.gov/pubmed/32906751
http://dx.doi.org/10.3390/molecules25184085

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7570916/
https://www.ncbi.nlm.nih.gov/pubmed/32906751
http://dx.doi.org/10.3390/molecules25184085

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