The matricellular protein CCN5 prevents adverse atrial structural and electrical remodelling

Atrial structural remodelling including atrial hypertrophy and fibrosis is a key mediator of atrial fibrillation (AF). We previously demonstrated that the matricellular protein CCN5 elicits anti‐fibrotic and anti‐hypertrophic effects in left ventricles under pressure overload. We here determined the...

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Detalles Bibliográficos
Autores principales: Lee, Min‐Ah, Raad, Nour, Song, Min Ho, Yoo, Jimeen, Lee, Miyoung, Jang, Seung Pil, Kwak, Tae Hwan, Kook, Hyun, Choi, Eun‐Kyoung, Cha, Tae‐Joon, Hajjar, Roger J., Jeong, Dongtak, Park, Woo Jin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2020
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7579720/
https://www.ncbi.nlm.nih.gov/pubmed/32885578
http://dx.doi.org/10.1111/jcmm.15789

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7579720/
https://www.ncbi.nlm.nih.gov/pubmed/32885578
http://dx.doi.org/10.1111/jcmm.15789

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