A Bibenzyl Component Moscatilin Mitigates Glycation-Mediated Damages in an SH-SY5Y Cell Model of Neurodegenerative Diseases through AMPK Activation and RAGE/NF-κB Pathway Suppression

Moscatilin can protect rat pheochromocytoma cells against methylglyoxal-induced damage. Elimination of the effect of advanced glycation end-products (AGEs) but activation of AMP-activated protein kinase (AMPK) are the potential therapeutic targets for the neurodegenerative diseases. Our study aimed...

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Detalles Bibliográficos
Autores principales: Lai, Mei Chou, Liu, Wayne Young, Liou, Shorong-Shii, Liu, I-Min
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7582731/
https://www.ncbi.nlm.nih.gov/pubmed/33036367
http://dx.doi.org/10.3390/molecules25194574

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7582731/
https://www.ncbi.nlm.nih.gov/pubmed/33036367
http://dx.doi.org/10.3390/molecules25194574

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