Development of experimental chronic kidney disease and vascular calcification alters diurnal variation of phosphate and its hormonal regulators

The mineral‐bone axis is tightly regulated and dependent on renal function. In chronic kidney disease (CKD) progressive loss of renal capacity disrupts this axis over‐time, with marked changes in circulating calcium, phosphate, PTH, and fibroblast growth factor‐23 (FGF‐23). These changes contribute...

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Detalles Bibliográficos
Autores principales: Svajger, Bruno A., Riddoch, Justin L. H., Pruss, Cynthia M., Laverty, Kimberly J., Ward, Emilie, Holden, Rachel M., Adams, Michael A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2020
Materias:
Original Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7666773/
https://www.ncbi.nlm.nih.gov/pubmed/33190417
http://dx.doi.org/10.14814/phy2.14626

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7666773/
https://www.ncbi.nlm.nih.gov/pubmed/33190417
http://dx.doi.org/10.14814/phy2.14626

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