Amyloid-β Peptide Impact on Synaptic Function and Neuroepigenetic Gene Control Reveal New Therapeutic Strategies for Alzheimer’s Disease

Amyloid-β (Aβ) peptides can form protease-resistant aggregates within and outside of neurons. Accumulation of these aggregates is a hallmark of Alzheimer’s disease (AD) neuropathology and contributes to devastating cognitive deficits associated with this disorder. The primary etiological factor for...

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Detalles Bibliográficos
Autores principales: Karisetty, Bhanu Chandra, Bhatnagar, Akanksha, Armour, Ellen M., Beaver, Mariah, Zhang, Haolin, Elefant, Felice
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2020
Materias:
Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7693454/
https://www.ncbi.nlm.nih.gov/pubmed/33304239
http://dx.doi.org/10.3389/fnmol.2020.577622

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7693454/
https://www.ncbi.nlm.nih.gov/pubmed/33304239
http://dx.doi.org/10.3389/fnmol.2020.577622

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