NOD1/RIP2 signalling enhances the microglia-driven inflammatory response and undergoes crosstalk with inflammatory cytokines to exacerbate brain damage following intracerebral haemorrhage in mice

BACKGROUND: Secondary brain damage caused by the innate immune response and subsequent proinflammatory factor production is a major factor contributing to the high mortality of intracerebral haemorrhage (ICH). Nucleotide-binding oligomerization domain 1 (NOD1)/receptor-interacting protein 2 (RIP2) s...

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Detalles Bibliográficos
Autores principales: Wang, Miao, Ye, Xinchun, Hu, Jinxia, Zhao, Qiuchen, Lv, Bingchen, Ma, Weijing, Wang, Weiwei, Yin, Hanhan, Hao, Qi, Zhou, Chao, Zhang, Tao, Wu, Weifeng, Wang, Yan, Zhou, Mingyue, Zhang, Cong-hui, Cui, Guiyun
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2020
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7708246/
https://www.ncbi.nlm.nih.gov/pubmed/33261639
http://dx.doi.org/10.1186/s12974-020-02015-9

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7708246/
https://www.ncbi.nlm.nih.gov/pubmed/33261639
http://dx.doi.org/10.1186/s12974-020-02015-9

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