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SKIL facilitates tumorigenesis and immune escape of NSCLC via upregulating TAZ/autophagy axis
Immune escape is an important mechanism in tumorigenesis. The aim of this study was to investigate roles of SKIL in tumorigenesis and immune escape of non-small-cell lung cancer (NSCLC). SKIL expression levels in NSCLC cell line, clinical sample, and adjacent normal tissue were measured by quantitat...
Autores principales: | , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7710697/ https://www.ncbi.nlm.nih.gov/pubmed/33268765 http://dx.doi.org/10.1038/s41419-020-03200-7 |