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The HLTF–PARP1 interaction in the progression and stability of damaged replication forks caused by methyl methanesulfonate
Human HLTF participates in the lesion-bypass mechanism through the fork reversal structure, known as template switching of post-replication repair. However, the mechanism by which HLTF promotes the replication progression and fork stability of damaged forks remains unclear. Here, we identify a novel...
Autores principales: | , , , , , , , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group UK
2020
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7719709/ https://www.ncbi.nlm.nih.gov/pubmed/33281189 http://dx.doi.org/10.1038/s41389-020-00289-5 |