Potential roles of neuronal nitric oxide synthase and the PTEN-induced kinase 1 (PINK1)/Parkin pathway for mitochondrial protein degradation in disuse-induced soleus muscle atrophy in adult rats

Excessive nitric oxide (NO) production and mitochondrial dysfunction can activate protein degradation in disuse-induced skeletal muscle atrophy. However, the increase in NO production in atrophied muscles remains controversial. In addition, although several studies have investigated the PTEN-induced...

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Detalles Bibliográficos
Autores principales: Uda, Munehiro, Yoshihara, Toshinori, Ichinoseki-Sekine, Noriko, Baba, Takeshi, Yoshioka, Toshitada
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2020
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7725317/
https://www.ncbi.nlm.nih.gov/pubmed/33296434
http://dx.doi.org/10.1371/journal.pone.0243660

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7725317/
https://www.ncbi.nlm.nih.gov/pubmed/33296434
http://dx.doi.org/10.1371/journal.pone.0243660

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