Hdac3 deletion in myeloid progenitor cells enhances bone healing in females and limits osteoclast fusion via Pmepa1

Previous studies examining the role of the histone deacetylase Hdac3 within myeloid cells demonstrated that Hdac3 promotes M2 activation and tissue healing in inflammatory conditions. Since myeloid lineage cells are required for proper bone formation and regeneration, in this study we examined the f...

Descripción completa

Detalles Bibliográficos
Autores principales: Molstad, David H. H., Zars, Elizabeth, Norton, Andrew, Mansky, Kim C., Westendorf, Jennifer J., Bradley, Elizabeth W.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2020
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7733476/
https://www.ncbi.nlm.nih.gov/pubmed/33311522
http://dx.doi.org/10.1038/s41598-020-78364-5

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7733476/
https://www.ncbi.nlm.nih.gov/pubmed/33311522
http://dx.doi.org/10.1038/s41598-020-78364-5

Ejemplares similares