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ENaC expression correlates with the acute furosemide‐induced K(+) excretion

BACKGROUND: In the aldosterone‐sensitive distal nephron (ASDN), epithelial sodium channel (ENaC)‐mediated Na(+) absorption drives K(+) excretion. K(+) excretion depends on the delivery of Na(+) to the ASDN and molecularly activated ENaC. Furosemide is known as a K(+) wasting diuretic as it greatly e...

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Detalles Bibliográficos
Autores principales: Ayasse, Niklas, Berg, Peder, Leipziger, Jens, Sørensen, Mads Vaarby
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7788322/
https://www.ncbi.nlm.nih.gov/pubmed/33410279
http://dx.doi.org/10.14814/phy2.14668