Emodin regulates cell cycle of non-small lung cancer (NSCLC) cells through hyaluronan synthase 2 (HA2)-HA-CD44/receptor for hyaluronic acid-mediated motility (RHAMM) interaction-dependent signaling pathway

BACKGROUND: Non-small cell lung cancers (NSCLC) account for most cases of lung cancer. More effort is needed to research new drug and combination therapies for this disease. An anthraquinone derivative, emodin shows anticancer potency. We hypothesis that emodin suppresses lung cancer cells through h...

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Detalles Bibliográficos
Autores principales: Li, Mingzhu, Jin, Shengbo, Cao, Yang, Xu, Jian, Zhu, Shendong, Li, Zheng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2021
Materias:
Primary Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7789699/
https://www.ncbi.nlm.nih.gov/pubmed/33407495
http://dx.doi.org/10.1186/s12935-020-01711-z

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7789699/
https://www.ncbi.nlm.nih.gov/pubmed/33407495
http://dx.doi.org/10.1186/s12935-020-01711-z

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