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Emodin regulates cell cycle of non-small lung cancer (NSCLC) cells through hyaluronan synthase 2 (HA2)-HA-CD44/receptor for hyaluronic acid-mediated motility (RHAMM) interaction-dependent signaling pathway
BACKGROUND: Non-small cell lung cancers (NSCLC) account for most cases of lung cancer. More effort is needed to research new drug and combination therapies for this disease. An anthraquinone derivative, emodin shows anticancer potency. We hypothesis that emodin suppresses lung cancer cells through h...
Autores principales: | Li, Mingzhu, Jin, Shengbo, Cao, Yang, Xu, Jian, Zhu, Shendong, Li, Zheng |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2021
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7789699/ https://www.ncbi.nlm.nih.gov/pubmed/33407495 http://dx.doi.org/10.1186/s12935-020-01711-z |
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