Cargando…
Insulin Receptor Substrate p53 Ameliorates High-Glucose-Induced Activation of NF-κB and Impaired Mobility of HUVECs
Diabetes-related macrovascular and microvascular complications lead to poor prognosis. Insulin receptor substrate p53 (IRSp53) is known to act as a substrate for the insulin receptor tyrosine kinase, but its role in endothelial dysfunction remains unclear. Human umbilical vein endothelial cells (HUV...
Autores principales: | Liu, Fen, Chen, Yubin, Zhao, Shi, Li, Mei, Luo, Fanyan, Tang, Can-e |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Hindawi
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7806382/ https://www.ncbi.nlm.nih.gov/pubmed/33506012 http://dx.doi.org/10.1155/2021/3210586 |
Ejemplares similares
-
Omentin-1 Ameliorated Free Fatty Acid-Induced Impairment in Proliferation, Migration, and Inflammatory States of HUVECs
por: Chen, Yubin, et al.
Publicado: (2020) -
Curcumin activates autophagy and attenuates high glucose-induced apoptosis in HUVECs through the ROS/NF-κB signaling pathway
por: Jin, Qi-Hui, et al.
Publicado: (2022) -
Artemisinin inhibits monocyte adhesion to HUVECs through the NF-κB and MAPK pathways in vitro
por: WANG, YUE, et al.
Publicado: (2016) -
Up-regulation of DcR3 in microbial toxins-stimulated HUVECs involves NF-κB signalling
por: Hou, Yanqiang, et al.
Publicado: (2018) -
Isoquercitrin protects HUVECs against high glucose-induced apoptosis through regulating p53 proteasomal degradation
por: Liu, Libo, et al.
Publicado: (2021)