Cardiac fibroblast miR‐27a may function as an endogenous anti‐fibrotic by negatively regulating Early Growth Response Protein 3 (EGR3)

Pathological myocardial fibrosis and hypertrophy occur due to chronic cardiac stress. The microRNA‐27a (miR‐27a) regulates collagen production across diverse cell types and organs to inhibit fibrosis and could constitute an important therapeutic avenue. However, its impact on hypertrophy and cardiac...

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Detalles Bibliográficos
Autores principales: Teng, Lifeng, Huang, Yubing, Guo, Jun, Li, Bin, Lin, Jin, Ma, Lining, Wang, Yudai, Ye, Cong, Chen, Qianqian
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2020
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7810947/
https://www.ncbi.nlm.nih.gov/pubmed/33215816
http://dx.doi.org/10.1111/jcmm.15814

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7810947/
https://www.ncbi.nlm.nih.gov/pubmed/33215816
http://dx.doi.org/10.1111/jcmm.15814

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