Advanced glycation end-products reduce lipopolysaccharide uptake by macrophages

Hyperglycaemia provides a suitable environment for infections and the mechanisms of glucose toxicity include the formation of advanced glycation end-products (AGEs), which comprise non-enzymatically glycosylated proteins, lipids, and nucleic acid amino groups. Among AGE-associated phenotypes, glycol...

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Detalles Bibliográficos
Autores principales: Kitaura, Atsuhiro, Nishinaka, Takashi, Hamasaki, Shinichi, Hatipoglu, Omer Faruk, Wake, Hidenori, Nishibori, Masahiro, Mori, Shuji, Nakao, Shinichi, Takahashi, Hideo
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2021
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7833212/
https://www.ncbi.nlm.nih.gov/pubmed/33493233
http://dx.doi.org/10.1371/journal.pone.0245957

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7833212/
https://www.ncbi.nlm.nih.gov/pubmed/33493233
http://dx.doi.org/10.1371/journal.pone.0245957

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