SAM68 promotes tumorigenesis in lung adenocarcinoma by regulating metabolic conversion via PKM alternative splicing

Background: A metabolic “switch” from oxidative phosphorylation to glycolysis provides tumor cells with energy and biosynthetic substrates, thereby promoting tumorigenesis and malignant progression. However, the mechanisms controlling this metabolic switch in tumors is not entirely clear. Methods: C...

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Detalles Bibliográficos
Autores principales: Zhu, Song, Chen, Weiping, Wang, Jizhong, Qi, Ling, Pan, Huilin, Feng, Zhengfu, Tian, Dongbo
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Ivyspring International Publisher 2021
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7847678/
https://www.ncbi.nlm.nih.gov/pubmed/33537092
http://dx.doi.org/10.7150/thno.51360

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7847678/
https://www.ncbi.nlm.nih.gov/pubmed/33537092
http://dx.doi.org/10.7150/thno.51360

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