Thioredoxin 1 is upregulated in the bone and bone marrow following experimental myocardial infarction: evidence for a remote organ response

Ischemia and reperfusion events, such as myocardial infarction (MI), are reported to induce remote organ damage severely compromising patient outcomes. Tissue survival and functional restoration relies on the activation of endogenous redox regulatory systems such as the oxidoreductases of the thiore...

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Detalles Bibliográficos
Autores principales: Godoy, José R., Pittrich, Sarah, Slavic, Svetlana, Lillig, Christopher Horst, Hanschmann, Eva-Maria, Erben, Reinhold G.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2020
Materias:
Original Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7847876/
https://www.ncbi.nlm.nih.gov/pubmed/33161477
http://dx.doi.org/10.1007/s00418-020-01939-w

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7847876/
https://www.ncbi.nlm.nih.gov/pubmed/33161477
http://dx.doi.org/10.1007/s00418-020-01939-w

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