RGC-32 Regulates Generation of Reactive Astrocytes in Experimental Autoimmune Encephalomyelitis

Astrocytes are increasingly recognized as critical contributors to multiple sclerosis pathogenesis. We have previously shown that lack of Response Gene to Complement 32 (RGC-32) alters astrocyte morphology in the spinal cord at the peak of experimental autoimmune encephalomyelitis (EAE), suggesting...

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Detalles Bibliográficos
Autores principales: Tatomir, Alexandru, Beltrand, Austin, Nguyen, Vinh, Boodhoo, Dallas, Mekala, Armugam, Cudrici, Cornelia, Badea, Tudor C., Muresanu, Dafin F., Rus, Violeta, Rus, Horea
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Immunology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7868332/
https://www.ncbi.nlm.nih.gov/pubmed/33569054
http://dx.doi.org/10.3389/fimmu.2020.608294

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7868332/
https://www.ncbi.nlm.nih.gov/pubmed/33569054
http://dx.doi.org/10.3389/fimmu.2020.608294

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