Cargando…
The IL33 receptor ST2 contributes to mechanical hypersensitivity in mice with neuropathic pain
Pathogen infection triggers pain via activation of the innate immune system. Toll-like receptors (TLRs) and Nod-like receptors (NLRs) are the main components of innate immunity and have been implicated in pain signaling. We previously revealed that the TLR2-NLRP3-IL33 pathway mediates inflammatory p...
Autores principales: | Huang, Junting, Gadotti, Vinicius M., Zhang, Zizhen, Zamponi, Gerald W. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7888111/ https://www.ncbi.nlm.nih.gov/pubmed/33596932 http://dx.doi.org/10.1186/s13041-021-00752-3 |
Ejemplares similares
-
Analgesic effects of optogenetic inhibition of basolateral amygdala inputs into the prefrontal cortex in nerve injured female mice
por: Gadotti, Vinicius M., et al.
Publicado: (2019) -
Disrupting USP5/Cav3.2 interactions protects female mice from mechanical hypersensitivity during peripheral inflammation
por: Gadotti, Vinicius M., et al.
Publicado: (2018) -
Inhibitory insula-ACC projections modulate affective but not sensory aspects of neuropathic pain
por: Alonso-Matielo, Heloísa, et al.
Publicado: (2023) -
Anxiolytic effects of the flavonoid luteolin in a mouse model of acute colitis
por: Gadotti, Vinicius M., et al.
Publicado: (2019) -
Analgesia by intrathecal delta-9-tetrahydrocannabinol is dependent on Cav3.2 calcium channels
por: Gadotti, Vinicius de Maria, et al.
Publicado: (2023)