TrkB signaling regulates the cold-shock protein RBM3-mediated neuroprotection

Increasing levels of the cold-shock protein, RNA-binding motif 3 (RBM3), either through cooling or by ectopic over-expression, prevents synapse and neuronal loss in mouse models of neurodegeneration. To exploit this process therapeutically requires an understanding of mechanisms controlling cold-ind...

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Detalles Bibliográficos
Autores principales: Peretti, Diego, Smith, Heather L, Verity, Nicholas, Humoud, Ibrahim, de Weerd, Lis, Swinden, Dean P, Hayes, Joseph, Mallucci, Giovanna R
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Life Science Alliance LLC 2021
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7893816/
https://www.ncbi.nlm.nih.gov/pubmed/33563652
http://dx.doi.org/10.26508/lsa.202000884

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7893816/
https://www.ncbi.nlm.nih.gov/pubmed/33563652
http://dx.doi.org/10.26508/lsa.202000884

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