Cargando…
Interleukin-1β mediates alterations in mitochondrial fusion/fission proteins and memory impairment induced by amyloid-β oligomers
BACKGROUND: The lack of effective treatments for Alzheimer’s disease (AD) reflects an incomplete understanding of disease mechanisms. Alterations in proteins involved in mitochondrial dynamics, an essential process for mitochondrial integrity and function, have been reported in AD brains. Impaired m...
Autores principales: | Batista, Andre F., Rody, Tayná, Forny-Germano, Leticia, Cerdeiro, Suzana, Bellio, Maria, Ferreira, Sergio T., Munoz, Douglas P., De Felice, Fernanda G. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2021
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7897381/ https://www.ncbi.nlm.nih.gov/pubmed/33612100 http://dx.doi.org/10.1186/s12974-021-02099-x |
Ejemplares similares
-
The Toxicity of Amyloid β Oligomers
por: Zhao, Li Na, et al.
Publicado: (2012) -
Development of new fusion proteins for visualizing amyloid-β oligomers in vivo
por: Ochiishi, Tomoyo, et al.
Publicado: (2016) -
Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer’s disease
por: Ferreira, Sergio T., et al.
Publicado: (2015) -
Breaking the Code of Amyloid-β Oligomers
por: Lesné, Sylvain E.
Publicado: (2013) -
The Toxicity and Polymorphism of β-Amyloid Oligomers
por: Huang, Ya-ru, et al.
Publicado: (2020)