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SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling

Glioblastoma (GBM) is the most common and aggressive brain tumor in adults. Overexpression or activation of epidermal growth factor receptor (EGFR) occurs commonly in multiple human cancers and promotes tumorigenesis. However, the underlying molecular mechanism of EGFR aberrant activation and the do...

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Autores principales: Song, Hai, Wang, Yanpei, Shi, Chaojia, Lu, Jianxiang, Yuan, Tian, Wang, Xiangpeng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7905166/
https://www.ncbi.nlm.nih.gov/pubmed/33643898
http://dx.doi.org/10.3389/fonc.2020.583984
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author Song, Hai
Wang, Yanpei
Shi, Chaojia
Lu, Jianxiang
Yuan, Tian
Wang, Xiangpeng
author_facet Song, Hai
Wang, Yanpei
Shi, Chaojia
Lu, Jianxiang
Yuan, Tian
Wang, Xiangpeng
author_sort Song, Hai
collection PubMed
description Glioblastoma (GBM) is the most common and aggressive brain tumor in adults. Overexpression or activation of epidermal growth factor receptor (EGFR) occurs commonly in multiple human cancers and promotes tumorigenesis. However, the underlying molecular mechanism of EGFR aberrant activation and the downstream signaling pathways remains largely unknown. In this study, we report that both SH3-domain kinase binding protein 1 (SH3KBP1) mRNA and protein levels are highly expressed in GBM and its high expression is associated with worse survival of glioma patients. In addition, we provide evidence that SH3KBP1 is prominently expressed in GBM stem cells (GSCs) and have potential to serve as a novel GSCs marker. Moreover, silencing SH3KBP1 dramatically impairs GBM cell proliferation, migration and GSCs self-renewal ability in vitro and xenograft tumors growth in vivo. Most importantly, we found that SH3KBP1 directly interacts with EGFR and may act as an adaptor protein to transduce EGFR signaling. Together, our work uncovers SH3KBP1 as a novel regulator of oncogenic EGFR signaling and also as a potential therapeutic target for GBM patients with EGFR activation.
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spelling pubmed-79051662021-02-26 SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling Song, Hai Wang, Yanpei Shi, Chaojia Lu, Jianxiang Yuan, Tian Wang, Xiangpeng Front Oncol Oncology Glioblastoma (GBM) is the most common and aggressive brain tumor in adults. Overexpression or activation of epidermal growth factor receptor (EGFR) occurs commonly in multiple human cancers and promotes tumorigenesis. However, the underlying molecular mechanism of EGFR aberrant activation and the downstream signaling pathways remains largely unknown. In this study, we report that both SH3-domain kinase binding protein 1 (SH3KBP1) mRNA and protein levels are highly expressed in GBM and its high expression is associated with worse survival of glioma patients. In addition, we provide evidence that SH3KBP1 is prominently expressed in GBM stem cells (GSCs) and have potential to serve as a novel GSCs marker. Moreover, silencing SH3KBP1 dramatically impairs GBM cell proliferation, migration and GSCs self-renewal ability in vitro and xenograft tumors growth in vivo. Most importantly, we found that SH3KBP1 directly interacts with EGFR and may act as an adaptor protein to transduce EGFR signaling. Together, our work uncovers SH3KBP1 as a novel regulator of oncogenic EGFR signaling and also as a potential therapeutic target for GBM patients with EGFR activation. Frontiers Media S.A. 2021-02-11 /pmc/articles/PMC7905166/ /pubmed/33643898 http://dx.doi.org/10.3389/fonc.2020.583984 Text en Copyright © 2021 Song, Wang, Shi, Lu, Yuan and Wang http://creativecommons.org/licenses/by/4.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
spellingShingle Oncology
Song, Hai
Wang, Yanpei
Shi, Chaojia
Lu, Jianxiang
Yuan, Tian
Wang, Xiangpeng
SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title_full SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title_fullStr SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title_full_unstemmed SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title_short SH3KBP1 Promotes Glioblastoma Tumorigenesis by Activating EGFR Signaling
title_sort sh3kbp1 promotes glioblastoma tumorigenesis by activating egfr signaling
topic Oncology
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7905166/
https://www.ncbi.nlm.nih.gov/pubmed/33643898
http://dx.doi.org/10.3389/fonc.2020.583984
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