Peptide Szeto-Schiller 31 ameliorates doxorubicin-induced cardiotoxicity by inhibiting the activation of the p38 MAPK signaling pathway

Oxidative stress serves a key role in doxorubicin (DOX)-induced cardiotoxicity. The peptide Szeto-Schiller (SS)31 is an efficacious antioxidant with the capacity to reduce mitochondrial reactive oxygen species (ROS) levels and scavenge free radicals. Although SS31 is involved in the pathophysiologic...

Descripción completa

Detalles Bibliográficos
Autores principales: Zhang, Li, Feng, Mengwen, Wang, Xuejun, Zhang, Hao, Ding, Jingjing, Cheng, Zijie, Qian, Lingmei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2021
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7914074/
https://www.ncbi.nlm.nih.gov/pubmed/33649779
http://dx.doi.org/10.3892/ijmm.2021.4896

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7914074/
https://www.ncbi.nlm.nih.gov/pubmed/33649779
http://dx.doi.org/10.3892/ijmm.2021.4896

Ejemplares similares