Cardiomyocyte depolarization triggers NOS-dependent NO transient after calcium release, reducing the subsequent calcium transient

Cardiac excitation–contraction coupling and metabolic and signaling activities are centrally modulated by nitric oxide (NO), which is produced by one of three NO synthases (NOSs). Despite the significant role of NO in cardiac Ca(2+) homeostasis regulation under different pathophysiological condition...

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Detalles Bibliográficos
Autores principales: Mosqueira, Matias, Konietzny, Roland, Andresen, Carolin, Wang, Chao, H.A. Fink, Rainer
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer Berlin Heidelberg 2021
Materias:
Original Contribution
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7966140/
https://www.ncbi.nlm.nih.gov/pubmed/33728868
http://dx.doi.org/10.1007/s00395-021-00860-0

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7966140/
https://www.ncbi.nlm.nih.gov/pubmed/33728868
http://dx.doi.org/10.1007/s00395-021-00860-0

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