Adropin inhibits the phenotypic modulation and proliferation of vascular smooth muscle cells during neointimal hyperplasia by activating the AMPK/ACC signaling pathway

In-stent restenosis (ISR) remains an inevitable problem for some patients receiving drug-eluting stent (DES) implantation. Intimal hyperplasia is an important biological cause of ISR. It has been previously reported that adropin is a potentially protective factor in cardiovascular disease. Therefore...

Descripción completa

Detalles Bibliográficos
Autores principales: Wang, Li, Zhao, Liang-Ping, Chen, Yu-Qi, Chang, Xian-Song, Xiong, Hui, Zhang, Dai-Min, Xu, Wei-Ting, Chen, Jian-Chang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2021
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8027754/
https://www.ncbi.nlm.nih.gov/pubmed/33850532
http://dx.doi.org/10.3892/etm.2021.9992

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8027754/
https://www.ncbi.nlm.nih.gov/pubmed/33850532
http://dx.doi.org/10.3892/etm.2021.9992

Ejemplares similares