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Adropin inhibits the phenotypic modulation and proliferation of vascular smooth muscle cells during neointimal hyperplasia by activating the AMPK/ACC signaling pathway

In-stent restenosis (ISR) remains an inevitable problem for some patients receiving drug-eluting stent (DES) implantation. Intimal hyperplasia is an important biological cause of ISR. It has been previously reported that adropin is a potentially protective factor in cardiovascular disease. Therefore...

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Detalles Bibliográficos
Autores principales:	Wang, Li, Zhao, Liang-Ping, Chen, Yu-Qi, Chang, Xian-Song, Xiong, Hui, Zhang, Dai-Min, Xu, Wei-Ting, Chen, Jian-Chang
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	D.A. Spandidos 2021
Materias:	Articles
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8027754/ https://www.ncbi.nlm.nih.gov/pubmed/33850532 http://dx.doi.org/10.3892/etm.2021.9992

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