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Lin28a ameliorates glucotoxicity-induced β-cell dysfunction and apoptosis

An excessive and prolonged increase in glucose levels causes β-cell dysregulation, which is accompanied by impaired insulin synthesis and secretion, a condition known as glucotoxicity. Although it is known that both Lin28a and Lin28b regulate glucose metabolism, other molecular mechanisms that may p...

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Detalles Bibliográficos
Autores principales: Hwang, Yeo Jin, Jung, Gwon-Soo, Jeon, WonBae, Lee, Kyeong-Min
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Korean Society for Biochemistry and Molecular Biology 2021
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8093937/
https://www.ncbi.nlm.nih.gov/pubmed/33691905
http://dx.doi.org/10.5483/BMBRep.2021.54.4.255

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