Nitric oxide down-regulates voltage-gated Na(+) channel in cardiomyocytes possibly through S-nitrosylation-mediated signaling

Nitric oxide (NO) is produced from endothelial cells and cardiomyocytes composing the myocardium and benefits cardiac function through both vascular-dependent and—independent effects. This study was purposed to investigate the possible adverse effect of NO focusing on the voltage-gated Na(+) channel...

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Detalles Bibliográficos
Autores principales: Wang, Pu, Wei, Mengyan, Zhu, Xiufang, Liu, Yangong, Yoshimura, Kenshi, Zheng, Mingqi, Liu, Gang, Kume, Shinichiro, Morishima, Masaki, Kurokawa, Tatsuki, Ono, Katsushige
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2021
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8163867/
https://www.ncbi.nlm.nih.gov/pubmed/34050231
http://dx.doi.org/10.1038/s41598-021-90840-0

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8163867/
https://www.ncbi.nlm.nih.gov/pubmed/34050231
http://dx.doi.org/10.1038/s41598-021-90840-0

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