Hydrogen Sulfide Attenuated Sepsis-Induced Myocardial Dysfunction Through TLR4 Pathway and Endoplasmic Reticulum Stress

Aims: We examined the change in endogenous hydrogen sulfide (H(2)S) production and its role in sepsis-induced myocardial dysfunction (SIMD). Results: Significant elevations in plasma cardiac troponin I (cTnI), creatine kinase (CK), tumor necrosis factor-α (TNF-α), and interleukin-1β (IL-1β) were not...

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Detalles Bibliográficos
Autores principales: Chen, Yu-hong, Teng, Xu, Hu, Zhen-jie, Tian, Dan-yang, Jin, Sheng, Wu, Yu-ming
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2021
Materias:
Physiology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8220204/
https://www.ncbi.nlm.nih.gov/pubmed/34177611
http://dx.doi.org/10.3389/fphys.2021.653601

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8220204/
https://www.ncbi.nlm.nih.gov/pubmed/34177611
http://dx.doi.org/10.3389/fphys.2021.653601

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